Face to Pubis delivery : What are the risk factors for it?

Medical Notes: Face to Pubis delivery : What are the risk factors for it?


Table of Contents(toc)

Which type of pelvis is associated with an increased incidence of face-to-pubis delivery?

a. Gynecoid pelvis
b. Anthropoid pelvis ✅ (Correct Answer)
c. Android pelvis
d. Platypelloid pelvis

Answer: b. Anthropoid pelvis


Solution:

Face-to-pubis delivery is most commonly associated with the Anthropoid pelvis.


Types of Pelvis:

1. Gynecoid Pelvis

  • Normal female pelvis

  • Most common type

  • Transverse diameter > Anteroposterior (AP) diameter

2. Anthropoid Pelvis

  • AP diameter > Transverse diameter

  • Face-to-pubis delivery is common

3. Android Pelvis

  • Male-type or wedge-shaped pelvis

  • All diameters reduced

  • Delayed engagement

  • Deep transverse arrest

4. Platypelloid Pelvis

  • Flat pelvis type

  • Transverse diameter > AP diameter

  • Least common type

  • Shortest diameter of pelvic inlet = Obstetric conjugate


Additional Important Points:

  • Most important diameter during labor: Interspinal diameter (of the pelvic outlet)

  • Shortest diameter of the fetal skull: Bimastoid diameter

  • Largest diameter of the fetal skull: Mento-vertical diameter

AFP Notes and MCQ (past questions)

AFP Notes and MCQ (past questions)


Table of Contents(toc)

Increase in Alpha-fetoprotein (AFP) is seen in:

a. Hepatoblastoma ✅ (Correct Answer)
b. Neuroblastoma
c. Thymoma
d. Angiosarcoma

Answer: a. Hepatoblastoma


Solution:

  • Alpha-fetoprotein (AFP) is a glycoprotein synthesized by the fetal yolk sac, fetal gastrointestinal tract, and fetal liver.

  • AFP concentration increases steadily until about 13 weeks of gestation (peak concentration in fetal plasma) and then gradually declines.

  • Peak maternal serum AFP occurs at 30–32 weeks of pregnancy.


Causes of Increased AFP:

1. Liver tumors:

  • Hepatoblastoma

  • Hepatocellular carcinoma

2. Germ cell tumors:

  • Yolk sac tumor (also known as Endodermal sinus tumor)

3. Others:

  • Neuroblastoma (Note: Can be associated but less specific compared to hepatoblastoma)


Causes of Increased Maternal Serum AFP (MSAFP):

Mnemonic: TAF-AFP

  • T: Thin pregnancy

  • A: Anencephaly

  • F: Fetal anomalies (e.g., open neural tube defects, abdominal wall defects)

Detailed list:

  • Miscalculation of gestational age (most common cause)

  • Open Neural Tube Defects (NTDs):

    • Anencephaly

    • Encephalocele

    • Spina bifida

  • Abdominal wall defects:

    • Gastroschisis

    • Omphalocele

  • Upper GI obstructions

  • Renal anomalies

  • Cystic hygroma

  • Pilonidal sinus

  • Ectodermal dysplasia

  • Aplasia cutis

  • Cystic adenomatoid malformation of the lungs

  • Multiple gestations


Causes of Decreased Amniotic Fluid AFP (AF-AFP):

  • Hydatidiform mole

  • Trisomy 21 (Down syndrome)

  • Trisomy 18 (Edwards syndrome)

  • Trisomy 13 (Patau syndrome)

  • Thin pregnancy

  • Misdated pregnancy

  • Missed abortion

Notes on Rheumatoid Arthritis for Medical Students in Short

Rheumatoid Arthritis

Table of Contents(toc)


Pathological Change

  • In Rheumatoid Arthritis (RA), the synovium becomes edematous (↑ fluid content, ↓ viscosity), leading to:

    • Thickening, then hypertrophy, and finally

    • Pannus formationhallmark of disease involving fibroblasts and small blood vessels.


General Characteristics

  • RA is a non-suppurative chronic disease.

  • Symmetrical inflammation of peripheral joints with progressive destruction of periarticular structures.

  • Associated with HLA Class II genes: HLA-DR, HLA-DQ.


Most Common Joints Affected

  • MCP of index finger > Wrist > PIP > MTP > Knee, Ankle, Shoulder, Elbow.

  • DIP joints spared (DIP joints are involved in Osteoarthritis, not RA).


Important Autoantibodies

  • Most specific: Anti-CCP antibodies.

  • Rheumatoid factor (RF): IgM antibodies directed against Fc portion of IgG.


Common Complications

  • Tendon rupture: Extensor digitorum communis (most common).

  • Eye complication: Dry eye (Sicca syndrome) — seen in ~38%.

  • Lung complication: Interstitial Lung Disease (ILD).


Characteristic Deformities

  • Z-deviation of hand:

    • Radial deviation at the wrist.

    • Ulnar deviation of fingers.

  • Swan neck deformity: Hyperextension of PIP, flexion of DIP.

  • Hill’s hiker’s thumb:

    • Extension at 1st IP joint.

    • Flexion at 1st MCP joint.

    • Loss of thumb mobility and pinch.

  • Hammer toes.


X-ray Findings

  • Juxta-articular osteopenia.

  • Bone erosions.

  • ↓ Joint space due to cartilage loss.


Syndromic Associations

  • Felty’s syndrome: Chronic RA + Splenomegaly + Neutropenia (<1500 cells/mm³).

  • Caplan’s syndrome: Pulmonary nodules + Pneumoconiosis in RA patients.


Treatment

  • First-line (DOC): Methotrexate (DMARD).

  • Best analgesic: Naproxen.

  • Biologics:

    • TNF inhibitors: Etanercept, Infliximab, Adalimumab.

    • Rituximab (anti-CD20 monoclonal antibody).


MCQ

Q. Which structure in the joint is affected earliest in Rheumatoid Arthritis?

a. Capsule
b. Articular cartilage
c. Synovium
d. Subchondral bone

Does hypothyroidism increase LDL Cholesterol, Here is New Fact about Your heart and thyroid?

Hypothyroidism and lipid profiyare closely related. Here’s ansito your question:

Yes, hypothyroidism can increase LDL (low-density lipoprotein) cholesterol levels.

Here’s why:

Thyroid hormones (mainly T3) help regulate lipid metabolism.
In hypothyroidism, low thyroid hormone levels lead to reduced LDL receptor activity in the liver.
This decreases the clearance of LDL from the bloodstream, causing elevated LDL levels.
It may also lead to increased total cholesterol and triglycerides, particularly in overt hypothyroidism.
Even subclinical hypothyroidism (normal T3/T4 but elevated TSH) can mildly increase LDL cholesterol.

Clinical relevance: 

In patients with high LDL or dyslipidemia, checking thyroid function is recommended, as treating hypothyroidism often improves lipid profiles.

antiemetic medications that can be used for nausea and vomiting in pregnancy (NVP)

Here’s a list of antiemetic medications that can be used for nausea and vomiting in pregnancy (NVP), typically in order of preference and safety:

First-line

1. Pyridoxine (Vitamin B6)
2. Doxylamine + Pyridoxine (e.g., Diclegis, Bonjesta) – FDA-approved combo for NVP

Second-line

3. Promethazine – Antihistamine with sedative effects
4. Dimenhydrinate – Antihistamine, often used for motion sickness
5. Diphenhydramine – Antihistamine, also helps with sleep
6. Meclizine – Another antihistamine safe in pregnancy

Third-line

7. Metoclopramide – Prokinetic agent; can cause extrapyramidal side effects
8. Ondansetron (Zofran) – Commonly used, some controversy over first trimester use but still widely prescribed

Others (used cautiously or in severe cases)

9. Prochlorperazine – Dopamine antagonist
10. Chlorpromazine – Older phenothiazine; more side effects
11. Trimethobenzamide – Less commonly used
Always tailor treatment to severity (mild, moderate, severe/Hyperemesis Gravidarum), and consider hydration/nutrition support if severe.

CBIMNCI protocol Nepal latest guideline free download

CBIMNCI (CB IMNCI) protocol Nepal for Health professionals

CBIMNCI protocol has been one of the driving guideline for treatment of neonatal and childhood illnesses in Nepal.

Here we have uploaded the latest CBIMNCI protocol book in pdf format for your reference. 

You can either refer it here or please download for future reference.

Download CBIMNCI (CB IMNCI) protocol

Community based integrated management of childhood and neonatal illness protocol has been developed by government of Nepal, child health division. This protocol has been the main guidelines for Nepal.

There are these protocols and guidelines developed for health care workers in Nepal.

  1. CB-IMNCI (CBIMNCI) guidelines for FCHV pdf
  2. FB-IMNCI (CBIMNCI) guidelines for Paramedics pdf
  3. FB-IMNCI (CBIMNCI) guidelines for Medical officers pdf
  4. CB IMNCI flipchart pdf
  5. CBIMNCI remote area  guideline pdf
  6. CBIMNCI program management module pdf

The complete guidelines can be downloaded below.

CBIMNCI remote area  guideline pdf (imnci chart booklet)

Here i CBIMNCI remote area guideline in pdf format to download and to read.

https://drive.google.com/file/d/1WMK9YeiYR0dIDPU9zaPvNEhZdRS2h5g0

CBIMNCI program management module pdf (imnci chart booklet)

Here is CBIMNCI program management module.

https://drive.google.com/file/d/11JiJMVooK7RVd2ER7O6yA5OMWdiOOF7O/preview

CB IMNCI flipchart pdf (imnci chart booklet)

Here is CBIMNCI flipchart for you to read and download.

https://drive.google.com/file/d/1cCVNF0ysFCYSYcKbwPCAlF8ZJ-cW1agS/preview

FB-IMNCI (CBIMNCI) guidelines for Paramedics pdf (imnci chart booklet)

https://drive.google.com/file/d/1z6XL030saQUPqacVJ4EKi5Euk7RwVDOj/preview

FB-IMNCI (CBIMNCI) guidelines for Medical officers pdf (imnci chart booklet)

https://drive.google.com/file/d/1S0ji6gsJyeSvbgdsV1hqVabhiSMkTkIj/preview

CB-IMNCI (CBIMNCI) guidelines for FCHV pdf (imnci chart booklet)

https://drive.google.com/file/d/1JE6Olq1Ko1oa8gs3jlpkEQeNq0_y9foA/preview

Download CBIMNCI (CB IMNCI) guideline (imnci chart booklet)

Please use this link for downloading the book in pdf format. These guidelines are updated accordingly to date of this post and updates will be posted subsequently.

Difference between CBIMNCI (CB IMNCI) and CB-IMCI

CBIMCI stands for community based integrated management of childhood illnesses. This guideline is the past form of CBIMNCI.

CBIMNCI was developed with addition of neonatal illnesses guidelines to the childhood illnesses. The main reason for this was that childhood illnesses cannot be separated from neonatal illnesses.

CBIMCI was also derived from IMCI (read below).

Definition of IMCI

IMCI stands for integrated management of childhood illnesses. As the childhood mortality rate in our country were relatively high this must have been addressed quickly and in a economic way. For this, healthcare workers like CMA, HA and volunteers like FCHV needed to be trained and upgraded to manage childhood illnesses. 

Gradually, this protocol was update to change into CBIMCI and subsequently into CBIMNCI and FBIMNCI.

IMCI vs IMNCI

The main difference between IMCI and IMNCI is that IMNCI includes neonatal ilnesses. 

Lately, IMNCI is updated into FB-IMNCI (Facility based integrated management of childhood and neonatal illnesses )

Timeline of CBIMNCI Nepal

IMCI-CBIMNCI-FCHV-PARAMEDICS-MEDICAL OFFICER-FBIMNCI

(See Picture below)

What are disease covered by CBIMNCI nepal?

Following diseases are covered by CBIMNCI and FBIMNCI Nepal.

  1. Diarrhoea and AGE
  2. Fever
  3. Otitis media (ear infections)
  4. Vomiting and dehydration
  5. Feeding problems

Should doctors follow CBIMNCI protocol Nepal?

Yes. There is a separate FBIMNCI protocol for medical officers. 

The protocol is different for rural and urban area as well. 

When was latest CBIMNCI guideline updated in Nepal?

Latest guidelines was updated in 2021 AD. The exact date of patest protocol are mentioned in the guidelines books above. Please refer to pdfs above.

Is CBIMNCI Nepal approved by WHO?

YES . CBIMNCI guidelines are guided and developed with help of WHO.

These guidelines can also be downloaded by visiting WHO website. 

Where can I download CBIMNCI guideline Nepal?

Please download the guidelines from links above. 

If you want to download in bundle refer this link. 

  1. WHO
  2. Child health division
  3. Family welfare division
  4. Dr Health Rx
  5. Ministry of health and population 

What are the risks of tear gas used in crowd control? ( fact based scientific and medical point of view)

Table of Contents(toc)

Tear gas, commonly used in crowd control, consists of chemical irritants such as chloroacetophenone (CN), chlorobenzylidene malononitrile (CS), and dibenzoxazepine (CR). While classified as a “riot control agent,” its effects on human health can be severe, particularly with prolonged or high-concentration exposure. 

Here are key risks supported by scientific and medical literature:

1. Respiratory Effects

• Acute Respiratory Distress Syndrome (ARDS): High exposure can lead to severe lung injury and ARDS, particularly in individuals with pre-existing conditions such as asthma or COPD.

• Bronchoconstriction & Asthma Exacerbation: CS gas can trigger acute bronchospasm, posing a significant risk for asthmatics.

• Lung Damage & Chronic Bronchitis: Prolonged exposure may result in chemical pneumonitis and chronic respiratory symptoms (Karagama et al., 2003).

2. Ocular Injuries & Blindness

• Severe Eye Irritation: Tear gas causes lacrimation, conjunctivitis, corneal abrasions, and in some cases, permanent vision impairment (Hu et al., 1989).

• Risk of Secondary Trauma: People often rub their eyes vigorously, leading to corneal damage and infection.

3. Skin Burns & Chemical Dermatitis

• Blister Formation & Irritation: CS and CN can cause second-degree burns, dermatitis, and allergic skin reactions (CDC, 2018).

• Delayed Hypersensitivity Reactions: Some individuals develop long-term skin sensitivity to tear gas chemicals.

4. Neurological & Psychological Effects

• Seizures & Nerve Damage: Reports suggest potential neurotoxicity with repeated exposure, including seizures in susceptible individuals (Papirmeister et al., 1991).

• PTSD & Anxiety Disorders: Tear gas exposure during stressful events has been linked to increased rates of PTSD, panic attacks, and acute stress reactions (Chorley et al., 2021).

5. Cardiovascular Risks

• Hypertension & Cardiac Events: Tear gas can increase blood pressure and heart rate, posing a risk for individuals with heart disease (Schep et al., 2015).

• Increased Risk of Heart Attacks: The sympathetic nervous system activation triggered by tear gas can induce myocardial infarction in vulnerable individuals.

6. Reproductive & Developmental Toxicity

• Increased Miscarriage Risk: Exposure to tear gas has been associated with miscarriages and menstrual irregularities in some studies (Karam et al., 2020).

• Potential Teratogenic Effects: Although data is limited, animal studies suggest possible fetal toxicity with prolonged exposure.

7. Long-Term Pulmonary & Systemic Effects

• Pulmonary Fibrosis & Chronic Lung Disease: Persistent exposure may lead to lung fibrosis, similar to occupational chemical exposures (Weisenburger et al., 2020).

• Potential Carcinogenicity: Some solvents used in tear gas formulations have been linked to DNA damage, though direct human studies are lacking.

Conclusion

Tear gas is not a harmless deterrent; it poses significant acute and chronic health risks, particularly for vulnerable populations (children, elderly, and those with pre-existing conditions). Its use in enclosed spaces or at high concentrations greatly increases risks of severe respiratory, ocular, and systemic effects.

Model MCQs for Nepal Government 8th Level Medical Officer Exam (NEW)

Model MCQs for Nepal Government 8th Level Medical Officer Exam

General Medicine 

  1. Which of the following is NOT a common cause of Chronic Obstructive Pulmonary Disease (COPD)?
    a) Smoking
    b) Air pollution
    c) Alpha-1 antitrypsin deficiency
    d) Tuberculosis
  2. A patient presents with fever, murmur, and Janeway lesions. What is the most likely diagnosis?
    a) Myocardial infarction
    b) Rheumatic heart disease
    c) Infective endocarditis
    d) Pericarditis
  3. Which investigation is most specific for diagnosing Hepatitis B infection?
    a) ALT/AST ratio
    b) HBsAg
    c) Anti-HBc IgM
    d) Anti-HAV IgM
  4. What is the most common type of anemia worldwide?
    a) Megaloblastic anemia
    b) Iron deficiency anemia
    c) Aplastic anemia
    d) Sickle cell anemia
  5. Which neurological condition is characterized by resting tremor, rigidity, and bradykinesia?
    a) Multiple sclerosis
    b) Parkinson’s disease
    c) Myasthenia gravis
    d) Guillain-Barré syndrome

    General Surgery

  6. The most common cause of acute appendicitis is:
    a) Fecalith obstruction
    b) Intestinal tuberculosis
    c) Volvulus
    d) Hernia
  7. Which is a common complication of deep vein thrombosis (DVT)?
    a) Pulmonary embolism
    b) Myocardial infarction
    c) Stroke
    d) Peripheral artery disease
  8. The first-line management for pneumothorax in a hemodynamically stable patient is:
    a) Chest tube insertion
    b) Needle decompression
    c) Oxygen therapy
    d) Observation
  9. Which condition is characterized by sudden onset of severe scrotal pain and absent cremasteric reflex?
    a) Testicular torsion
    b) Epididymitis
    c) Hydrocele
    d) Varicocele
  10. Which of the following is the best initial investigation for breast carcinoma?
    a) Mammography
    b) FNAC
    c) MRI
    d) Ultrasound

     Obstetrics and Gynecology

  11. A patient at 32 weeks gestation presents with painless vaginal bleeding. The most likely diagnosis is:
    a) Placenta previa
    b) Abruptio placentae
    c) Ectopic pregnancy
    d) Uterine rupture
  12. Which of the following is NOT a risk factor for ectopic pregnancy?
    a) Pelvic inflammatory disease
    b) Previous ectopic pregnancy
    c) In vitro fertilization
    d) Multiparity

Answer Key:

  1. d) Tuberculosis
  2. c) Infective endocarditis
  3. b) HBsAg
  4. b) Iron deficiency anemia
  5. b) Parkinson’s disease
  6. a) Fecalith obstruction
  7. a) Pulmonary embolism
  8. c) Oxygen therapy
  9. a) Testicular torsion
  10. a) Mammography
  11. a) Placenta previa
  12. d) Multiparity

Nonspecific Urethritis (NSU) – Notes (PSC Focused)

Definition:

Nonspecific urethritis (NSU) is urethral inflammation not caused by Neisseria gonorrhoeae. It is usually due to other bacterial, viral, or non-infectious causes. This article has PSC Nepal focused notes on Nonspecific urethritis (NSU).

Etiology:

  1. Infectious Causes:
  2. Chlamydia trachomatis (most common)
  3. Mycoplasma genitalium
  4. Ureaplasma urealyticum
  5. Trichomonas vaginalis
  6. Herpes simplex virus (HSV)
  7. Adenoviruses
  8. Non-infectious Causes:
  9. Chemical irritants (e.g., soaps, spermicides)
  10. Trauma (e.g., catheterization, vigorous sexual activity)

Risk Factors:

  1. Unprotected sexual intercourse
  2. Multiple sexual partners
  3. History of sexually transmitted infections (STIs)
  4. Poor genital hygiene

Clinical Features:

  1. Urethral discharge (clear, mucoid, or purulent)
  2. Dysuria (burning sensation while urinating)
  3. Urethral pruritus or discomfort
  4. Possible hematuria (rare)
  5. Symptoms may be mild or asymptomatic in some cases

Diagnosis:

  1. Clinical Diagnosis: Based on symptoms and exclusion of gonorrhea
  2. Laboratory Tests:
  3. Urinalysis (pyuria without bacteriuria)
  4. Gram stain of urethral discharge (absence of intracellular diplococci)
  5. Nucleic Acid Amplification Test (NAAT) for Chlamydia trachomatis and Mycoplasma genitalium
  6. Culture for Ureaplasma and Mycoplasma (if available)

Differential Diagnosis:

  1. Gonococcal urethritis
  2. Prostatitis
  3. Cystitis
  4. Epididymitis

Management:

  1. Empirical Antibiotic Therapy:
  2. First-line:
  3. Azithromycin 1g single dose OR
  4. Doxycycline 100mg BID for 7 days
  5. Alternative:
  6. Moxifloxacin 400mg daily for 7-14 days (if M. genitalium is suspected and resistant to doxycycline)
  7. Adjunct Measures:
  8. Avoid sexual intercourse until symptoms resolve and treatment is completed
  9. Partner notification and treatment (to prevent reinfection)
  10. Follow-up in 2-4 weeks if symptoms persist

Complications:

  1. Chronic urethritis
  2. Epididymitis
  3. Reactive arthritis (Reiter’s syndrome)
  4. Infertility (rare)

Prevention:

  1. Safe sexual practices (consistent condom use)
  2. Regular STI screening
  3. Avoidance of potential chemical irritants

Erb’s Palsy vs Klumpke’s Paralysis

Q. Erb’s Palsy:

a. Occurs typically usually after breech delivery of smaller babies
b. Is due to injury of C5 and C6 nerve roots
c. All fingers muscles are paralysed
d. There may be unilateral Horner’s syndrome
Ans: ‘b’

Solution

Explanation: Injury to upper trunk of Brachial plexus (C5, C6, C7) leads t60 Erb’s palsy.

Erb’s palsy

❖ Brachial plexus may be injured when person falls from a height on the side of head and shoulder whereby the nerves of the plexus are violently stretched. (upper trunk of the plexus injured).

Clinical features of Erb’s Palsy

Paralysis of Ms Deltoid, biceps, brachialis, intraspinatus and spinator.
The position of limb is characteristic i.e., the arm hanges by the side medially rotated and the forearm is extended and pronated (Policeman’s tip) 
  • Weakness of proximal upper limb muscles 
  • Loss of sensation in area supplied by c5,6,7 dermatome distribution 

Klumpke’s Paralysis 

Cause (pathoanatomy) of Klumpke’s Paralysis 

Klumpke’s paralysis caused by injury in lower trunk of brachial plexus (C8, T1) characterized by paralysis of intrinsic hand Ms & C8/T1 dermatome distribution numbness.

Clinical features of Klumpke’s Paralysis 

Clinically Klumpke’s Paralysis has following features 
  • Weakness of distal muscle of upper limbs
  • Wasting of forearm muscles
  • Flexion of wrist (wrist drop)
  • Clawing of hand
  • Numbing/loss of sensation of c8 and t1 distribution 
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